This hypothesis is supported when we observe that in the highest dose, the extract is able to favor the production of IFN-γ, demonstrating a clear tendency for the Th1 response, where the activation of the cellular immune response allows the destruction of tumor cells through mechanisms dependent on (CD8 +) and independent of (NK, NKT) the presentation of the MHC class I [50,51]. This evidence concerns the gene IFNG and neoplasm.