However, some noteworthy strengths of this study are the inclusions of different non-AD tauopathies (e.g., PSP, PiD, and GGT, as well as the two different clinical variants of PSP [PSP-RS and PSP-SL] associated with relatively different ratios of cortical to subcortical 4R tau. This evidence concerns the gene MTA2 and supranuclear palsy, progressive, 1.