IL4 and psoriasis: The expression of AMPs such as cathelicidin and some β-defensins is partially inhibited on lesional skin of patients with atopic dermatitis and is much less than that observed on Th17-dominant inflammatory responses such as wounding or psoriasis.10 The suppression of these AMPs occurs in part through the action of interleukin-4 (IL-4) and IL-13.11,12 An over-growth of other species of Staphylococci, in particular S. epidermidis,2,13,14 is also seen in Th2 inflammation.