STMN1 and cervical cancer: In conclusion, propofol at high concentrations enhanced the cytotoxicity of paclitaxel, resulting in reduced malignancy of cervical cancer cells, by downregulating stathmin 1 expression, while propofol at clinically relevant concentrations had no effect on the efficacy of paclitaxel in cervical cancer cells; therefore, propofol at clinically relevant concentrations might not affect the response to paclitaxel in patients with recurrent or advanced cervical cancer.