MYC and hypertrophic cardiomyopathy: The results showed that prognostic differential genes mainly affected KEGG-ribosome, KEGG-focal adhesion, KEG-spliceosome, KEGG-DNA replication, KEGG-hypertrophic cardiomyopathy HCM and other pathways (Fig. 5C) and HALLMARK-E2F targets, HALLMARK-epithelial mesenchymal transition, HALLMARK-G2M checkpoint, HALLMARK-MYC targets V1, HALLMARK-myogenesis and other related functions (Fig. 5D).