This study revealed that the administration of DAB/PB for 2 months significantly increased the level of the hepatic MDA and NO and significantly decrease the antioxidant activity of SOD and Gpx; this agrees with Rajkapoor and his colleagues (Rajkapoor et al. 2005) who reported that the lipid peroxidation (LPO) can produce many of harmful byproducts, including 4-hydroxy nonenal and MDA, which can assault DNA and cause mutagenicity and cancer. The gene discussed is SOD1; the disease is cancer.