We discuss future therapeutic options aimed at inhibiting the pCRP‐LPC mediated dissociation associated with brain pathology, for example, compound 1,6‐bis‐PC, injected intravenously, prevented mCRP deposition and associated damage, after temporary left anterior descending artery ligation and myocardial infarction in a rat model. The gene discussed is PCSK7; the disease is myocardial infarction.