Numerous studies over the past few years have demonstrated that long noncoding RNAs (lncRNAs) can function as oncogenes[10, 11, 12, 13] or tumor suppressors[14, 15, 16] to participate in tumor progression and chemoresistance through a variety of mechanisms, such as epigenetic, transcriptional, and post‐transcriptional regulation.[17, 18, 19, 20, 21, 22] However, the lncRNAs that directly regulate c‐Src activity during cancer development remain largely unknown. Here, SRC is linked to neoplasm.