Mice deficient in Caspase-1, IL-1β, or IL-18 reportedly succumbed to Salmonella infection earlier than WT-mice, with significantly higher bacterial burdens in the spleens, Peyer’s patches, and mesenteric lymph nodes [15], indicating that inflammasome activation is a key target for Salmonella immune evasion strategies. This evidence concerns the gene IL1B and Salmonella Infections.