Other studies showed that OXT restored abnormal neuronal morphology and synaptic plasticity deficits in the medial prefrontal cortex and hippocampus; rescued attention, social recognition, and social memory deficits in Shank3-deficient rats, which were commonly used as animal models of ASD (Harony-Nicolas et al., 2017; Reichova et al., 2020); and improved or regulated social preference, social bond, social approach, and sexual behavior, as well as decreased stress response, anxiety, and aggression behavior in animals (Braida et al., 2012; Chang and Platt, 2014; Wei et al., 2020). Here, OXT is linked to Anxiety.