In M2 TAMs, stress granule (SG) formation was stimulated by tumor micro-environment (TME) stress, and SG increased DDX3Y/hnRNPF mediated mRNA stability of CCL13, which in turn enhanced CCL13 expression and promoted the metastasis of oral squamous cell carcinoma metastasis, however, the above molecular expression and phenotype are reversed upon knockdown of the G3BP1 (15). Here, CCL13 is linked to neoplasm.