UXT and viral infectious disease: UXT-V2 is phosphorylated by BGLF4 at the Thr3 site, which blocks the interaction between UXT-V2 and P65; this inhibits the transactivation of NF-κB, effectively weakening the immune response of host cells in the antiviral response, and facilitating the replication process of the virus (29).Therefore, UXT-V2 and UXT-V1 can regulate the innate immune response during viral infection.