The outcomes from that paper were that some immune cells, including F4/80- and CD11c-positive macrophages, granzyme B-positive NK cells/cytotoxic T lymphocytes, and IL-17–positive Th-17 cells, contribute to the pathogenesis of endometriosis by increasing the secretion of proinflammatory cytokines (58). This evidence concerns the gene GZMB and endometriosis.