Alzheimer's disease (AD) is characterized by the accumulation of β-amyloid (Aβ) peptides and hyperphosphorylated tau (p-Tau) protein in the form of senile plaques and neurofibrillary tangles, respectively, which cause neurotoxicity and neuronal loss leading to memory and learning impairments [5–7]. This evidence concerns the gene MAPT and Alzheimer disease.