TBCE and hydrops fetalis: MiR-155 was reported to be involved in high fat-high cholesterol-high sugar (HF-HC-HS) diet-induced steatosis and liver fibrosis, as miR-155 knockout mice showed significantly less liver injury, decreased steatosis, and attenuation in fibrosis under HF-HC-HS diet or CCl4 treatment, and KCs isolated from miR-155 KO mice displayed the M2 phenotype when exposed to even M1 priming conditions (116, 117).