Furthermore, some studies have found that elevated expression of the various Wnt-related proteins, including nuclear β-catenin, WNT3A, and cyclin D1, in nasal polyp tissues could be accompanied by the overexpression of mesenchymal markers, thus suggesting that Wnt signaling might be involved in the pathogenesis of nasal polyp formation via stimulation of the activation of EMT (80). The gene discussed is CCND1; the disease is nasal cavity polyp.