The lack of long-term care facilities [16] (where the virus spread especially in the first pandemic wave), the climate, [14] people’s tendency to spend time outdoors [17] but also genetic variants of the human angiotensin-converting enzyme-2 (ACE2) and the transmembrane serine protease 2 (TMPRSS2) receptors [18] and effective public health emergency mitigation strategies (already experienced during the Ebola outbreak) [16] were invoked to account for the phenomenon. This evidence concerns the gene ACE2 and Ebola hemorrhagic fever.