Under the experimental conditions, the mRNA levels of antiapoptotic Bcl2 and proapoptotic Gsk3b are altered in similar ways, i.e., both hypoxia and ischemia significantly increased their levels (for Bcl2, 1.48-fold increase in hypoxia and 1.64-fold increase in ischemia; for Gsk3b, 1.29- and 1.36-fold increase in hypoxia and ischemia, respectively). This evidence concerns the gene BCL2 and ischemia.