The transcription factor SP1 served as the inspiration for the overexpression of LINC00511 in glioma cells. Additionally, the upregulation of LINC00511 competitively sponges the miR-124-3p, driving the production of CCND2 and the cyclin D2 protein produced by this gene, which may hold significant potential for glioma therapies. Here, CCND2 is linked to glioma.