The percentage of myeloid-derived (neutrophils, monocytes, macrophages, dendritic cells) nor lymphoid cells (NK cells, B cells, T cells) between control and myeloid HIF-2α-deficient tumors using the E0771 orthotopic murine breast tumor model were also unchanged when tumor burden became significantly different (~13 days) nor at end point (~16 days/1.2 cm) [59]. The gene discussed is EPAS1; the disease is breast neoplasm.