Amyloidosis may be caused by deposition of an intrinsically abnormal protein (e.g., hereditary transthyretin [hATTR] amyloidosis and acquired systemic immunoglobulin light-chain [AL] amyloidosis), prolonged exposure to excess of a normal protein (e.g., reactive systemic [AA] amyloidosis and β2-microglobulin dialysis-related amyloidosis), or by the ageing process (e.g., wild-type transthyretin amyloidosis [ATTRwt]). Here, HLA-G is linked to amyloidosis.