CD8A and COVID-19: Although there were no major differences in respect to inflammatory changes between patients who died of SARS-CoV2-related pneumonia and those with COVID-19 who died of other conditions, patients with longer intensive care and septic state had, in general, more perivascular CD8-positive cells and diffuse microglial activation than patients with shorter disease duration and acute death, caused by myocardial complications or the pulmonary embolism.