Although there were no major differences in respect to inflammatory changes between patients who died of SARS-CoV2-related pneumonia and those with COVID-19 who died of other conditions, patients with longer intensive care and septic state had, in general, more perivascular CD8-positive cells and diffuse microglial activation than patients with shorter disease duration and acute death, caused by myocardial complications or the pulmonary embolism. This evidence concerns the gene CD8A and susceptibility to pneumonia measurement.