Tepebaşı et al. [52] studied the effects of inflammation markers (neutrophil–lymphocyte ratio, serum reactive protein, procalcitonin, etc.), IL-10 and IF γ levels, and oxidative stress on DNA damage in 95 patients hospitalized with COVID-19, who were divided into three groups according to the severity of pneumonia—mild, moderate and severe/critical. The gene discussed is IL10; the disease is pneumonia.