It effectively reduces the growth and volume of tumors induced by ACS gastric cancer cells injected in nude mice, with the underlying mechanism being the induction of apoptosis, reactive oxygen species (ROS) formation, the loss of mitochondrial membrane potential (MMP), m-TOR/PI3K/AKT signaling inhibition and the withholding of cancer cell migration and invasion. The gene discussed is AKT1; the disease is cancer.