In a 2021 follow-up study designed to explore the corneal and vascular VK status in SCD patients by focusing on matrix MGP, Sarosiak et al. [98] reported for the first time, “quite unexpected and remarkable findings” related to MGP expression in human corneas, along with differential perturbations by SCD UBIAD1 variants of associated VK metabolic pathways. The gene discussed is UBIAD1; the disease is Schnyder corneal dystrophy.