Our results agree with reports stating that SARS-CoV-2-specific NK-cell-mediated antibody-dependent cell-mediated cytotoxicity (ADCC) responses were subjected to NK cell FcγRIIIa genetic variants [41] and that the normal activity of NK cells might improve the control of COVID-19 by fighting the virus and suppressing fibrosis progression [42,43]. The gene discussed is FCGR3A; the disease is COVID-19.