STING1 and metabolic dysfunction-associated steatotic liver disease: Since it has been reported that STING can induce proinflammatory mediator release [35,36,37] and is related to lipid metabolism [25,34] and changes in the intestinal microbiota [38], all of which are mechanisms involved in NAFLD progression, correlations between STING and inflammatory cytokines, liver enzymes, lipid metabolism-related genes, and hepatic Toll-like receptors and some microbiota-derived metabolites, such as bile acids, were determined.