SMARCA5 and posterior cortical atrophy: A study from Maitland et al. suggested that GR could increase the expression levels of miR-99a and miR-100, which would disrupt the stem cell phenotype of PCa by directly targeting SMARCA5 (an SWI/SNF-related matrix-associated actin-dependent regulator of chromatin subfamily A member 5) and SMARCD1 (an SWI/SNF-related matrix-associated actin-dependent regulator of chromatin subfamily D member 1) [165].