As shown in Figure 6 and Figure 7, both EOs (0.025% v/v) were able to significantly reduce TNF-α-induced NF-κB p65 cytoplasm–nucleus translocation in both C- and GDM-HUVEC, while in basal conditions, this effect reached statistical significance only in GDM-HUVEC pre-incubated with LEO (Figure 7b). The gene discussed is NFKB1; the disease is gestational diabetes.