Indeed, previously reported recurring pathogenic variants (PVs) amongst Druze include two PVs in the ATM gene (the gene that underlies Ataxia Telangectesia–OMIM # 208900) in Druze communities in Jordan, Lebanon, and Syria [4]; a PV in the β globin gene [5]; and a nonsense variant in the LDL receptor (LDLR) gene, causing familial hypercholesterolemia [6]. This evidence concerns the gene LDLR and familial hypercholesterolemia.