It was found to affect the MAPK pathway, which increases p53 expression and leads to the alteration of the cytosolic concentration of calcium; calcium acts as a second messenger, and its intracellular increase influences the proliferation and migration of glioblastoma cells by modulating the formyl peptide receptor-like 1 (FPRL-1) [41,130]. The gene discussed is FPR2; the disease is glioblastoma.