Our data are consistent with a previous report that showed that LPS led to decreased lncRNA TUG1 expression and enhanced miR‐153‐3p levels in HRMCs (Figure 2J,K), implying that lncRNA TUG1 and miR‐153‐3p are involved in LN in an in vitro model. The gene discussed is TUG1; the disease is lobular neoplasia.