IL1B and acute respiratory distress syndrome: Narciclasine treatment also attenuated pathological injury and pulmonary edema and suppressed the secretion of IL-6, IL-1β, TNF-α, and MCP-1 as well as reduced the expression of intercellular adhesion molecule-1 (ICAM-1) and vascular cell adhesion molecule 1 (VCAM-1) in neonatal ALI rats.