Given the large contribution of ILC3s to the production of IL-22 in the intestine, we next used conditional knockout animals for the aryl hydrocarbon receptor (Ahr) in the RORγt compartment (hereby named AhrΔRORγt) to investigate the effects of inulin diet in this classic murine model of ILC3 deficiency [88]. This evidence concerns the gene IL22 and hyperinsulinemic hypoglycemia, familial, 4.