A single case had an ETV6-NCOA2 fusion, a previously described driver of T/myeloid leukemia.17 Notably, all TAL1 cases co-occurred with either a LMO1 or LMO2 lesion, with LMO1 lesions significantly more frequent in IF than in responsive cases (10.4% v 1.5%; P = .006). This evidence concerns the gene TAL1 and myeloid leukemia.