The results of IF analysis of Ki‐67 in the transplanted tumor specimens from the animals indicated that mice in the sh‐1232#1 group expressed less Ki‐67 than mice in the sh‐NC group; next, LINC01232 knockdown and simultaneous overexpression of NBR1 could reverse this phenomenon (Figure S10a, Supporting Information). The gene discussed is NBR1; the disease is neoplasm.