VSNL1 and colorectal carcinoma: The present study showed that the ability of VSNL1 to suppress anticancer drug‐induced apoptosis depended on myristoylation, Ca2+‐binding, and protein dimerization, thus suggesting that the Ca2+‐myristoyl switch is indispensable for the antiapoptotic function of VSNL1 in CRC cells.