Whereas, SARS‐CoV‐2 induced a great inflammatory signature, as exemplified by high mRNA levels of typical proinflammatory cytokines like IL‐6, TNF‐α, and IFN‐γ (Figure 1C), consistent with what has been observed in people with COVID‐19.[13] Next, we analyzed viral replication in respiratory tract organs, including turbinate, trachea, and lung, by RT‐PCR that amplifies SARS‐CoV‐2 open reading frame 1ab (ORF1ab) to detect viral RNA load in the homogenized tissues collected at 5 dpi. Here, IFNG is linked to COVID-19.