For instance, the microtubule‐disrupting agent paclitaxel can elicit an autophagic response that actually plays a protective role, impeding its antitumoral efficiency.[29, 30, 31] Consistently, we found that the expressions of autophagy‐related proteins, LC3, ATG5, and BECN1 were increased in TNBC tissues as compared to other breast cancer tissues, or as compared to the normal tissues, confirming activated autophagy existing in human TNBC specimens (Figure S5a,b, Supporting Information). This evidence concerns the gene ATG5 and breast carcinoma.