Very recently, a cohort study showed that acute COVID-19 or postacute sequelae of COVID-19 (PASC) are not related to autoantibodies but to elevated plasma levels of proinflammatory cytokines such as interleukin-1 beta (IL-1β), IL-6, and tumor necrosis factor alpha (TNF-α) [12]. The gene discussed is IL1B; the disease is COVID-19.