Of relevance, the lack of glucose receptors in the fetal brain (GLUT1 deficiency syndrome) (Gilbert-Jaramillo et al., 2019; Blonz, 2016) causes congenital microcephaly and fetal neurodevelopmental abnormalities; shared phenotypes with Zika congenital syndrome (Blonz, 2016; Wen et al., 2017; Merfeld et al., 2017; Mlakar et al., 2016). This evidence concerns the gene SLC2A1 and microcephaly.