In gastric adenocarcinoma with an intestinal mucin phenotype, most interactions were seen for Parvimonas, Sediminibacterium, Helicobacter, Selenomonas, Fusobacterium, Reyranella, Treponema, Leptotrichia, Haemophilus, Neisseria, Veillonella, Prevotella, and Streptococcus (Fig. 5B; Table S5). Here, MUC5AC is linked to gastric adenocarcinoma.