Upon activation, neutrophils release TNF‐α to induce DC maturation, enabling these DCs to trigger strong T cell immunity.[51] In line with this, we demonstrated that neutrophil–DC interactions, mediated by ligand–receptor pairs, facilitated more tumor antigen presentation to cytotoxic CD8+ T cells, resulting in antitumor immunity in HCC. The gene discussed is TNF; the disease is neoplasm.