STK4 and non-small cell lung carcinoma: In addition, G‐Rh2 inactivated Wnt signaling, upregulated STK4 expression, inhibited cell viability and cell cycle, and enhanced cell apoptosis, which was attenuated by miR‐28‐5p mimic in NSCLC cells, suggesting that G‐Rh2 exerted its anti‐proliferation effect by affecting miR‐28‐5p/STK4 axis and the following inactivation of Wnt signaling.