Previous studies have reported that some variants in crystallin genes (e.g., CRYBB1, CRYBB2, CRYAA, and CRYGC) could lead to a combined occurrence of CC and other ocular abnormalities, such as microcornea, microphthalmia, coloboma, and glaucoma [6, 8]. This evidence concerns the gene CRYBB2 and microphthalmia.