The physiological attenuation of the proinflammatory Th1 related cytokines, lasting until third trimester [55], can be a protective factor form extensive pulmonary damage and ARDS, as it was reported that IL-1β, IL-6, TNF-α are a good marker of cytokine response (and cytokine storm), with their lower serum levels associated with higher survival rates [77]. The gene discussed is IL6; the disease is acute respiratory distress syndrome.