As an additional OMM protein, we further analyzed the levels of the mitochondrial import receptor subunit TOM70 (Fig. 6a), which upon depolarization revealed a trend towards increased levels for the heterozygous carrier group compared to the control group (p = 0.101; 59.55 ± 12 vs. 31.03 ± 12.2 respectively), and a significant increase for the homozygous PRKN-PD patient neurons (p = 0.002), as compared to the controls (100.2 ± 14.1 vs. 31.03 ± 12.2) (Fig. 6c). This evidence concerns the gene PRKN and Parkinson disease.