MST1R and type 1 diabetes mellitus: The differences between DIO and T1D regarding the pathways associated with inflammatory responses (TREM1, PRR, MAPK, HMGB1, STAT3, Th1/Th2, and MSP-RON) and oxidative stress (production of NO and ROS, the antioxidant action of vitamin C) might be responsible for enhanced lung toxicity of PM exposure in T1D mice.