Further mechanism research revealed that NEAT1-1 could interact with cyclin L1 (CCNL1) through its #4 m6A and acted as a bridge between CCNL1 and cyclin dependent kinase 19 (CDK19) to form a stable complex, which may be specific in in bone metastatic prostate cancer. This evidence concerns the gene CDK19 and metastatic prostate carcinoma.