Notably, slc39a10 morphants developed severe anemia based on o‐dianisidine staining (Figure 1D,E), accompanied by a clear reduction in HSPCs measured using whole‐mount in situ hybridization (WISH) for cmyb (Figure 1F); in contrast, these developmental changes were not observed in slc39a6 morphants (Figure 1D,F). This evidence concerns the gene SLC39A6 and anemia.